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The mechanism by which mivacurium chloride exerts its action is most likely by binding competitively to cholinergic receptors on the motor end-plate to antagonise the action of acetylcholine, resulting in a block of neuromuscular transmission.↗TGA PIT1✓
“The plasma terminal elimination half-life varied between 1.74 and 2.63 minutes. In liver transplant patients, the plasma half-life of mivacurium was increased to 4.4 minutes compared to 1.75 minutes in subjects with normal liver function; the duration of paralysis was prolonged also.”